［1］李禹正, 张婷, 王晓民. 生物钟的秘密——2017年诺贝尔生理学或医学奖简介［J］. 首都医科大学学报, 2017, 38(5): 765－769.
［2］王可, 张婷, 王晓民. 大脑中的 “定位系统”——2014年诺贝尔生理学或医学奖简介［J］. 首都医科大学学报, 2014, 35(5): 671－675.
［3］汪璇, 王晓民. 细胞命运的逆转: 体细胞核移植与诱导多能干细胞——2012年诺贝尔生理学或医学奖简介［J］. 首都医科大学学报, 2012, 33(5): 689－694.
［4］张婷, 王晓民. 体外受精与试管婴儿——2010年诺贝尔生理学或医学奖简介［J］. 首都医科大学学报, 2010, 31(5): 678－682.
［5］杨宏彦, 王晓民. 由 “程序性细胞死亡” 透视2002年诺贝尔生理学或医学奖［J］. 生理通讯, 2002, 21(6): 178－181.
［6］王晓民. 攻克器官和组织移植的免疫排斥——1990年诺贝尔生理学或医学奖授与二位美国医生Murray和Thomas［J］. 生理科学进展, 1991, 22(2): 191.
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［13］Zhen J L, Qian Y J, Weng X C, et al. Gamma rhythm low field magnetic stimulation alleviates neuropathologic changes and rescues memory and cognitive impairments in a mouse model of Alzheimer's disease［J］. Alzheimers Dement (N Y), 2017, 3(4): 487－497.
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［16］Zhang T, Gong X L, Hu G Z, et al. EP2-PKA signaling is suppressed by triptolide in lipopolysaccharide-induced microglia activation［J］. J Neuroinflammation, 2015, 12: 50.
［17］Gong Y T, Xue B, Jiao J, et al. Triptolide inhibits COX-2 expression and PGE2 release by suppressing the activity of NF-κB and JNK in LPS-treated microglia［J］. J Neurochem, 2008, 107(3): 779－788.
［18］Zheng Y, Zhang W J, Wang X M. Triptolide with potential medicinal value for diseases of the central nervous system［J］. CNS Neurosci Ther, 2013, 19(2): 76－82.
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［20］Cui Y Q, Jia Y J, Zhang T, et al. Fucoidan protects against lipopolysaccharide-induced rat neuronal damage and inhibits the production of proinflammatory mediators in primary microglia［J］. CNS Neurosci Ther, 2012, 18(10): 827－833.
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［22］Yuan H L, Li B, Xu J, et al. Tenuigenin protects dopaminergic neurons from inflammation-mediated damage induced by the lipopolysaccharide［J］. CNS Neurosci Ther, 2012, 18(7): 584－590.
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［24］Wang J J, Niu D B, Zhang T, et al. A tetracycline-regulatable adeno-associated virus vector for double-gene transfer［J］. Neurosci Lett, 2005, 378(2): 106－110.
［25］Wang X, Wu H T, Zhang Z X, et al. Effects of interleukin-6, leukemia inhibitory factor, and ciliary neurotrophic factor on the proliferation and differentiation of adult human myoblasts［J］. Cell Mol Neurobiol, 2008, 28(1): 113－124.
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［29］Wang L, Gong X L, Liu Y, et al. CD200 maintains the region-specific phenotype of microglia in the midbrain and its role in Parkinson's disease［J］. Glia, 2020, 68(9): 1874－1890.
［30］Fan Z, Pan Y T, Zhang Z Y, et al. Systemic activation of NLRP3 inflammasome and plasma α-synuclein levels are correlated with motor severity and progression in Parkinson's disease［J］. J Neuroinflammation, 2020, 17(1): 11.
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［32］王晓民, 李林. 神经变性病教育部重点实验室的建设与发展［J］. 首都医科大学学报, 2010, 31(4): 497－508.
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［36］王晓民, 王韵, 王世强. 执着和奉献:纪念中国生理学会成立90周年:1926-2016 ［M］. 北京:高等教育出版社, 2016.
［37］王晓民. 我国科技计划的布局及脑重大疾病研究战略的思考［J］. 中国药理学与毒理学杂志, 2019, 33(6): 401.
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