［1］Yang J K, Feng Y, Yuan M Y, et al. Plasma glucose levels and diabetes are Independent predictors for mortality and morbidity in patients with SARS［J］. Diabet Med, 2006, 23(6): 623-628.
［2］Niu M J, Yang J K, Lin S S, et al. Loss of angiotensin-converting enzyme 2 leads to impaired glucose homeostasis in mice［J］. Endocrine, 2008, 34(1/3): 56-61.
［3］Yang J K, Lin S S, Ji X J, et al. Binding of SARS coronavirus to its receptor damages islets and causes acute diabetes［J］. Acta Diabetol, 2010, 47(3): 193-199.
［4］Yang J K, Zhou J B, Xin Z, et al. Interactions among related genes of renin-angiotensin system associated with type 2 diabetes［J］. Diabetes Care, 2010, 33(10): 2271-2273.
［5］Zhao M M, Zhu Y, Zhang L, et al. Novel cleavage sites identified in SARS-CoV-2 spike protein reveal mechanism for cathepsin L-facilitated viral infection and treatment strategies［J］. Cell Discov, 2022, 8(1): 53.
［6］Zhao M M, Yang W L, Yang F Y, et al. Cathepsin L plays a key role in SARS-CoV-2 infection in humans and humanized mice and is a promising target for new drug development［J］. Signal Transduct Target Ther, 2021, 6(1): 134.
［7］Cao X, Song L N, Yang J K. ACE2 and energy metabolism: the connection between COVID-19 and chronic metabolic disorders［J］. Clin Sci (Lond), 2021, 135(3): 535-554.
［8］Cao X, Lu X M, Tuo X, et al. Angiotensin-converting enzyme 2 regulates endoplasmic reticulum stress and mitochondrial function to preserve skeletal muscle lipid metabolism［J］. Lipids Health Dis, 2019, 18(1): 207.
［9］Cao X, Song L N, Zhang Y C, et al. Angiotensin-converting enzyme 2 inhibits endoplasmic reticulum stress-associated pathway to preserve nonalcoholic fatty liver disease［J］. Diabetes Metab Res Rev, 2019, 35(4): e3123.
［10］Shi T T, Yang F Y, Liu C, et al. Angiotensin-converting enzyme 2 regulates mitochondrial function in pancreatic β-cells［J］. Biochem Biophys Res Commun, 2018, 495(1): 860-866.
［11］Zhang F, Liu C, Wang L, et al. Antioxidant effect of angiotensin (1-7) in the protection of pancreatic β cell function［J］. Mol Med Rep, 2016, 14(3): 1963-1969.
［12］Cao X, Yang F Y, Shi T T, et al. Angiotensin-converting enzyme 2/angiotensin-(1-7)/Mas axis activates Akt signaling to ameliorate hepatic steatosis［J］. Sci Rep, 2016, 6: 21592.
［13］Cao X, Yang F Y, Xin Z, et al. The ACE2/Ang-(1-7)/Mas axis can inhibit hepatic insulin resistance［J］. Mol Cell Endocrinol, 2014, 393(1/2): 30-38.
［14］Liu C, Lv X H, Li H X, et al. Angiotensin-(1-7) suppresses oxidative stress and improves glucose uptake via Mas receptor in adipocytes［J］. Acta Diabetol, 2012, 49(4): 291-299.
［15］Yang J K, Lu J, Yuan S S, et al. From hyper- to hypoinsulinemia and diabetes: effect of KCNH6 on insulin secretion［J］. Cell Rep, 2018, 25(13): 3800-3810.e6.
［16］Zhao M M, Lu J, Li S, et al. Berberine is an insulin secretagogue targeting the KCNH6 potassium channel［J］. Nat Commun, 2021, 12(1): 5616.
［17］Lu J, Shen H, Li Q, et al. KCNH6 protects pancreatic β-cells from endoplasmic reticulum stress and apoptosis［J］. FASEB J, 2020, 34(11): 15015-15028.
［18］Lu J, Shen H, Li Q, et al. Effect of KCNH6 on hepatic endoplasmic reticulum stress and glucose metabolism［J］. Horm Metab Res, 2020, 52(9): 669-675.
［19］Lu J, Shi T T, Yuan S S, et al. Cisapride induced hypoglycemia via the KCNH6 potassium channel［J］. Front Endocrinol (Lausanne), 2022, 13: 1011238.
［20］Zhang Y C, Xiong F R, Cheng C, et al. KCNH6 enhanced hepatic glucose metabolism through mitochondrial Ca2+ regulation and oxidative stress inhibition［J］. Oxid Med Cell Longev, 2022, 2022: 3739556.
［21］Yang J K, Wang Y Y, Liu C, et al. Urine proteome specific for eye damage can predict kidney damage in patients with type 2 diabetes: a case-control and a 5.3-year prospective cohort study［J］. Diabetes Care, 2017, 40(2): 253-260.
［22］Zheng X M, Yang F Y, Chen X, et al. Development of a sensitive and reliable ELISA kit of urinary haptoglobin to predict progress of diabetic kidney disease［J］. Diabetes Metab Res Rev, 2021, 37(4): e3432.
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